Morgagni-Adams-Stokes syndrome: различия между версиями
Wikiadmin (обсуждение | вклад) (Новая страница: «en:Morgagni-Adams-Stokes syndrome ru:Синдром Морганьи — Адамса — Стокса <b>Morgagni-Adams-Stokes syndrome</b> is a syncope…») |
Wikiadmin (обсуждение | вклад) |
||
| Строка 5: | Строка 5: | ||
Morgagni-Adams-Stokes syndrome can occur in such forms: | Morgagni-Adams-Stokes syndrome can occur in such forms: | ||
| − | # Tachycardic: develops with paroxysmal ventricular tachycardia, paroxysm of supraventricular tachycardia and paroxysm of atrial fibrillation or [[atrial flutter]] at a ventricular contraction rate of more than 250 beats per minute, with [[Wolff-Parkinson-White syndrome (WPW syndrome)]]. | + | # Tachycardic: develops with paroxysmal ventricular tachycardia, paroxysm of supraventricular tachycardia and paroxysm of atrial fibrillation or [[atrial flutter]] at a ventricular contraction rate of more than 250 beats per minute, with the [[Wolff-Parkinson-White syndrome (WPW syndrome)]]. |
# Bradycardic: develops with the refusal or stopping of the sinus node, [[third-degree or complete atrioventricular block|complete atrioventricular block]] and [[sinoatrial block]] at a frequency of contractions of the ventricles 20 beats per minute or less. | # Bradycardic: develops with the refusal or stopping of the sinus node, [[third-degree or complete atrioventricular block|complete atrioventricular block]] and [[sinoatrial block]] at a frequency of contractions of the ventricles 20 beats per minute or less. | ||
# Mixed: develops with alternating periods of [[ventricular asystole]] and tachyarrhythmia. | # Mixed: develops with alternating periods of [[ventricular asystole]] and tachyarrhythmia. | ||
| − | Bouts usually occur suddenly, rarely last more than 1-2 minutes and, as a rule, do not entail neurological complications. Acute myocardial infarction or cerebral circulation disorders can be both a cause and a consequence of Morgagni-Adams-Stokes syndrome. | + | Bouts usually occur suddenly, rarely last more than 1-2 minutes and, as a rule, do not entail neurological complications. Acute myocardial infarction or cerebral circulation disorders can be both a cause and a consequence of the Morgagni-Adams-Stokes syndrome. |
The clinical picture: the patient suddenly loses consciousness, becomes pale with a dianetic tinge, the pupils are dilated and do not respond to light, the cervical veins swell, the breathing becomes deep, then rare, irregular; the muscles of the trunk become strained, lightly twitching of the muscles of the face and hands occur first, and then epileptiform convulsions occur; the pulse is not palpable, the heart sounds are not listened to, blood pressure is not determined. The termination of an bout is accompanied by a sharp reddening of the skin. An attack lasting 4-5 minutes can end fatal. Light attacks are characterized by a slight dizziness and blackout of consciousness, accompanied by a sharp pallor. It is often possible to determine the reason for bouts by outpatient monitoring. | The clinical picture: the patient suddenly loses consciousness, becomes pale with a dianetic tinge, the pupils are dilated and do not respond to light, the cervical veins swell, the breathing becomes deep, then rare, irregular; the muscles of the trunk become strained, lightly twitching of the muscles of the face and hands occur first, and then epileptiform convulsions occur; the pulse is not palpable, the heart sounds are not listened to, blood pressure is not determined. The termination of an bout is accompanied by a sharp reddening of the skin. An attack lasting 4-5 minutes can end fatal. Light attacks are characterized by a slight dizziness and blackout of consciousness, accompanied by a sharp pallor. It is often possible to determine the reason for bouts by outpatient monitoring. | ||
Версия 12:09, 25 марта 2017
Morgagni-Adams-Stokes syndrome is a syncope caused by a sharp decrease in cardiac output and cerebral ischemia due to an acute cardiac arrhythmia (second-degree sinoatrial block or complete atrioventricular block, paroxysmal tachycardia, ventricular fibrillation, sick sinus syndrome and etc.).
Morgagni-Adams-Stokes syndrome can occur in such forms:
- Tachycardic: develops with paroxysmal ventricular tachycardia, paroxysm of supraventricular tachycardia and paroxysm of atrial fibrillation or atrial flutter at a ventricular contraction rate of more than 250 beats per minute, with the Wolff-Parkinson-White syndrome (WPW syndrome).
- Bradycardic: develops with the refusal or stopping of the sinus node, complete atrioventricular block and sinoatrial block at a frequency of contractions of the ventricles 20 beats per minute or less.
- Mixed: develops with alternating periods of ventricular asystole and tachyarrhythmia.
Bouts usually occur suddenly, rarely last more than 1-2 minutes and, as a rule, do not entail neurological complications. Acute myocardial infarction or cerebral circulation disorders can be both a cause and a consequence of the Morgagni-Adams-Stokes syndrome.
The clinical picture: the patient suddenly loses consciousness, becomes pale with a dianetic tinge, the pupils are dilated and do not respond to light, the cervical veins swell, the breathing becomes deep, then rare, irregular; the muscles of the trunk become strained, lightly twitching of the muscles of the face and hands occur first, and then epileptiform convulsions occur; the pulse is not palpable, the heart sounds are not listened to, blood pressure is not determined. The termination of an bout is accompanied by a sharp reddening of the skin. An attack lasting 4-5 minutes can end fatal. Light attacks are characterized by a slight dizziness and blackout of consciousness, accompanied by a sharp pallor. It is often possible to determine the reason for bouts by outpatient monitoring.
Treatment:
If the cause of the syndrome is tachyarrhythmias, it is necessary to prescribe the appropriate antiarrhythmic drugs. If the bouts occur as a result of bradycardia (most often with complete atrioventricular block), a permanent electrocardiostimulation is advisable. The best method of treatment for this syndrome is implantation of the electrodes of the artificial pacemaker (electrostimulator) in the heart.
