Morgagni-Adams-Stokes syndrome: различия между версиями

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Morgagni-Adams-Stokes syndrome can occur in such forms:
 
Morgagni-Adams-Stokes syndrome can occur in such forms:
  
# Tachycardic: develops with paroxysmal ventricular tachycardia, paroxysm of supraventricular tachycardia and paroxysm of atrial fibrillation or [[atrial flutter]] at a ventricular contraction rate of more than 250 beats per minute, with [[Wolff-Parkinson-White syndrome (WPW syndrome)]].
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# Tachycardic: develops with paroxysmal ventricular tachycardia, paroxysm of supraventricular tachycardia and paroxysm of atrial fibrillation or [[atrial flutter]] at a ventricular contraction rate of more than 250 beats per minute, with the [[Wolff-Parkinson-White syndrome (WPW syndrome)]].
 
# Bradycardic: develops with the refusal or stopping of the sinus node, [[third-degree or complete atrioventricular block|complete atrioventricular block]] and [[sinoatrial block]] at a frequency of contractions of the ventricles 20 beats per minute or less.
 
# Bradycardic: develops with the refusal or stopping of the sinus node, [[third-degree or complete atrioventricular block|complete atrioventricular block]] and [[sinoatrial block]] at a frequency of contractions of the ventricles 20 beats per minute or less.
 
# Mixed: develops with alternating periods of [[ventricular asystole]] and tachyarrhythmia.
 
# Mixed: develops with alternating periods of [[ventricular asystole]] and tachyarrhythmia.
  
Bouts usually occur suddenly, rarely last more than 1-2 minutes and, as a rule, do not entail neurological complications. Acute myocardial infarction or cerebral circulation disorders can be both a cause and a consequence of Morgagni-Adams-Stokes syndrome.
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Bouts usually occur suddenly, rarely last more than 1-2 minutes and, as a rule, do not entail neurological complications. Acute myocardial infarction or cerebral circulation disorders can be both a cause and a consequence of the Morgagni-Adams-Stokes syndrome.
  
The clinical picture: the patient suddenly loses consciousness, becomes pale with a dianetic tinge, the pupils are dilated and do not respond to light, the cervical veins swell, the breathing becomes deep, then rare, irregular; the muscles of the trunk become strained, lightly twitching of the muscles of the face and hands occur first, and then epileptiform convulsions occur; the pulse is not palpable, the heart sounds are not listened to, blood pressure is not determined. The termination of an bout is accompanied by a sharp reddening of the skin. An attack lasting 4-5 minutes can end fatal. Light attacks are characterized by a slight dizziness and blackout of consciousness, accompanied by a sharp pallor. It is often possible to determine the reason for bouts by outpatient monitoring.
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The clinical picture: the patient suddenly loses consciousness, becomes pale with a dianetic tinge, the pupils are dilated and do not respond to light, the cervical veins swell, the breathing becomes deep, then rare, irregular; the muscles of the trunk become strained, lightly twitching of the muscles of the face and hands occur first, and then epileptiform convulsions occur; the pulse is not palpable, the heart sounds are not listened to, blood pressure is not determined. The termination of an bout is accompanied by a sharp reddening of the skin. An attack lasting 4-5 minutes can end fatal. Light attacks are characterized by a slight dizziness and blackout of consciousness, accompanied by a sharp pallor.  
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It is often possible to determine the reason for bouts by outpatient monitoring. At home, this can be done using the ECG Dongle [https://cardio-cloud.ru/].
  
 
<b>Treatment:</b>
 
<b>Treatment:</b>
  
 
If the cause of the syndrome is tachyarrhythmias, it is necessary to prescribe the appropriate antiarrhythmic drugs. If the bouts occur as a result of [[bradycardia]] (most often with [[third-degree or complete atrioventricular block|complete atrioventricular block]]), a permanent electrocardiostimulation is advisable. The best method of treatment for this syndrome is implantation of the electrodes of the artificial pacemaker (electrostimulator) in the heart.
 
If the cause of the syndrome is tachyarrhythmias, it is necessary to prescribe the appropriate antiarrhythmic drugs. If the bouts occur as a result of [[bradycardia]] (most often with [[third-degree or complete atrioventricular block|complete atrioventricular block]]), a permanent electrocardiostimulation is advisable. The best method of treatment for this syndrome is implantation of the electrodes of the artificial pacemaker (electrostimulator) in the heart.

Текущая версия на 10:46, 27 ноября 2020

Morgagni-Adams-Stokes syndrome is a syncope caused by a sharp decrease in cardiac output and cerebral ischemia due to an acute cardiac arrhythmia (second-degree sinoatrial block or complete atrioventricular block, paroxysmal tachycardia, ventricular fibrillation, sick sinus syndrome and etc.).

Morgagni-Adams-Stokes syndrome can occur in such forms:

  1. Tachycardic: develops with paroxysmal ventricular tachycardia, paroxysm of supraventricular tachycardia and paroxysm of atrial fibrillation or atrial flutter at a ventricular contraction rate of more than 250 beats per minute, with the Wolff-Parkinson-White syndrome (WPW syndrome).
  2. Bradycardic: develops with the refusal or stopping of the sinus node, complete atrioventricular block and sinoatrial block at a frequency of contractions of the ventricles 20 beats per minute or less.
  3. Mixed: develops with alternating periods of ventricular asystole and tachyarrhythmia.

Bouts usually occur suddenly, rarely last more than 1-2 minutes and, as a rule, do not entail neurological complications. Acute myocardial infarction or cerebral circulation disorders can be both a cause and a consequence of the Morgagni-Adams-Stokes syndrome.

The clinical picture: the patient suddenly loses consciousness, becomes pale with a dianetic tinge, the pupils are dilated and do not respond to light, the cervical veins swell, the breathing becomes deep, then rare, irregular; the muscles of the trunk become strained, lightly twitching of the muscles of the face and hands occur first, and then epileptiform convulsions occur; the pulse is not palpable, the heart sounds are not listened to, blood pressure is not determined. The termination of an bout is accompanied by a sharp reddening of the skin. An attack lasting 4-5 minutes can end fatal. Light attacks are characterized by a slight dizziness and blackout of consciousness, accompanied by a sharp pallor.

It is often possible to determine the reason for bouts by outpatient monitoring. At home, this can be done using the ECG Dongle [1].

Treatment:

If the cause of the syndrome is tachyarrhythmias, it is necessary to prescribe the appropriate antiarrhythmic drugs. If the bouts occur as a result of bradycardia (most often with complete atrioventricular block), a permanent electrocardiostimulation is advisable. The best method of treatment for this syndrome is implantation of the electrodes of the artificial pacemaker (electrostimulator) in the heart.